A novel drug has been created which prevented the blood clotting associated with heart attacks and strokes in pre-clinical studies.
List view / Grid view
Filter the results
The novel nanoparticle delivered Mp6 peptide was able to inhibit thrombosis and reduce inflammation without increasing the risk of bleeding in mice after heart attacks.
The 3D map, created using Knife-Edge Scanning Microscopy and analysed using laser capture microdissection, reveals new insights about the nervous control of the heart.
Researchers exploring how stem cells can be used discovered that cardiopoietic stem cells could repair in tissue in mouse models of heart attacks.
Research determined macrophages’ role in forming a permanent cardiac scar after a heart attack, the researchers suggest altering this behaviour may make the scar temporary and allow restoration of heart function.
Researchers have shown that a protein therapy has been successful in pre-clinical models at improving the quality of scar tissue after heart attacks, leading to better overall heart function.
A myocardial infarction model has been developed which will contribute to the development of preventive/therapeutic medicine.
A world-first compound that can keep cells alive and functioning in a healthy state could be revolutionary for medical emergencies.
Researchers have uncovered a treatment that may be used to treat heart attacks or to prevent heart failure, using the circadian clock.
A new target has been identified for the treatment of heart failure, heart attack, stroke and neurodegeneration.
This research advances the possibility of reviving the heart's regenerative capacities using microRNAs.
A novel antiplatelet therapy could help to prevent heart attacks and stroke through preventing the formation of blood clots...
A blood test that quickly and easily detects whether a person is at risk of a secondary heart attack is being developed by scientists at the Baker Heart and Diabetes Institute...
The new class of drugs are mechanism-based inhibitors which potentially interrupt the gut microbial pathway that produces TMAO...